FIRE AND EXPLOSION WITH HAZMAT EVACUATION AT CHEMICAL PLANT ON 6TH ST IN SEWARD, IL

    SUN JUN 02 2013 ~12:41 PM
    PECATONICA FPD
    BOX ALARM 2ND LEVEL/STRUCTURE FIRE WITH EXPLOSION
    16161 6TH STREET SEWARD, IL

    MABAS DIVISION 8 BOX 12 FOR STRUCTURE FIRE WITH EXPLOSIONS. MABAS DISPATCH STATE FACILITY IS "CHEM TOOL". MANDATORY EVACUATIONS UNDERWAY FOR POSSIBLE CHEMICAL EXPOSURE BELIEVED TO INVOLVE HEXACHLORODISILANE -- NOT CONFIRMED.



    MUTUAL AID: ENGINES: WIN-BUR-SEW, MOUNT MORRIS, WEST SUBURBAN, GERMAN VALLEY, BLACKHAWK, SOUTH BELOIT. TENDERS/TANKERS: BYRON, BOONE 3, LEAF RIVER, GERMAN VALLEY, NORTHWEST, FREEPORT RURAL, STILLMAN VALLEY. TRUCKS: BYRON, HARLEM-ROSCOE. SQUADS : NORTH PARK RIT, CHERRY VALLEY RIT. EMS: METRO, LIFELINE. CHIEFS: WIN-BUR-SEW, NORTH PARK, DURAND, CHERRY VALLEY, MABAS DUTY OFFICER. HAZMAT FOR SPECIAL ASSIGNMENT ON STANDBY.  MABAS 8 DECON.

    LEAF RIVER UNABLE TO RESPOND WITH TENDER.

    CHANGE OF QUARTERS: DURAND ENGINE, ORFORDVILLE TENDER, BLACKHAWK CHIEF, ATS MEDICAL.

    STAGING MOVED TO SEWARD PARKING LOT ONE BLOCK WEST OF THE SCENE AND THEN MOVED AT 1:29 PM TO SEWARD AG "NORTH OF THE TRACKS."

    WIND N 8-9 MPH WITH GUSTS TO 18. NATIONAL WEATHER SERVICE FORECASTS THE WIND TO SHIFT TO THE NORTHEAST AT ABOUT 9:00 PM AND TO THE EAST AT ABOUT MIDNIGHT, SOUTH AT 3:00 AM AND WEST AT ABOUT 5 AM.

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    HEXACHLORODISILANE INFO ...

    EMERGENCY OVERVIEW
    RISK
    Heating may cause an explosion.
    Reacts violently with water.
    Causes severe burns.
    Risk of serious damage to eyes.
    POTENTIAL HEALTH EFFECTS

    ACUTE HEALTH EFFECTS
    SWALLOWED

    The material can produce severe chemical burns within the oral cavity and gastrointestinal tract following ingestion.

    Ingestion of acidic corrosives may produce burns around and in the mouth. the throat and esophagus. Immediate pain and difficulties in
    swallowing and speaking may also be evident. Swelling of the epiglottis may make it difficult to breathe which may result in suffocation. More
    severe exposure may result in vomiting blood and thick mucus, shock, abnormally low blood pressure, fluctuating pulse, shallow respiration
    and clammy skin, inflammation of stomach wall, and rupture of esophageal tissue. Untreated shock may eventually result in kidney failure.
    Severe cases may result in perforation of the stomach and abdominal cavity with consequent infection, rigidity and fever. There may be
    severe narrowing of the esophageal or pyloric sphincters; this may occur immediately or after a delay of weeks to years. There may be coma
    and convulsions, followed by death due to infection of the abdominal cavity, kidneys or lungs.

    Ingestion of liquid chlorosilanes may cause severe internal injury or death. The chlorosilanes all react with moisture in the air or water to
    produce hydrogen chloride, the principal reaction product that can cause acute injury to any body tissue contacted.

    Ingestion of liquid chlorosilane could cause severe corrosive burns of the mouth, esophagus and stomach, potentially resulting in perforated
    viscus with subsequent chemical pleuritis, mediastinitis and peritonitis.

    The material has NOT been classified as "harmful by ingestion". This is because of the lack of corroborating animal or human evidence.
    The material may still be damaging to the health of the individual, following ingestion, especially where pre-existing organ (e.g. liver, kidney)
    damage is evident. Present definitions of harmful or toxic substances are generally based on doses producing mortality (death) rather than
    those producing morbidity (disease, ill-health). Gastrointestinal tract discomfort may produce nausea and vomiting. In an occupational setting
    however, unintentional ingestion is not thought to be cause for concern.
    EYE

    The material can produce severe chemical burns to the eye following direct contact. Vapors or mists may be extremely irritating.

    If applied to the eyes, this material causes severe eye damage.

    Direct eye contact with acid corrosives may produce pain, tears, sensitivity to light and burns. Mild burns of the epithelia generally recover
    rapidly and completely. Severe burns produce long-lasting and possibly irreversible damage. The appearance of the burn may not be
    apparent for several weeks after the initial contact. The cornea may ultimately become deeply opaque resulting in blindness.

    Direct contact of chlorosilane liquid with the eyes causes severe corrosive damage that may result in total loss of sight. Vapour exposure to
    the eyes may result in significant irritation up to severe chemical burns that may result in total loss of sight.

    Irritation of the eyes may produce a heavy secretion of tears (lachrymation).
    SKIN

    The material can produce severe chemical burns following direct contactwith the skin.

    Skin contact with acidic corrosives may result in pain and burns; these may be deep with distinct edges and may heal slowly with the
    formation of scar tissue.

    Skin contact is not thought to have harmful health effects, however the material may still produce health damage following entry through
    wounds, lesions or abrasions.

    Skin contact with either chlorosilane vapour or liquid can cause burns varying in severity from first to third degree, depending on
    concentration and length of exposure.

    Open cuts, abraded or irritated skin should not be exposed to this material.

    Solution of material in moisture on the skin, or perspiration, may markedly increase skin corrosion and accelerate tissue destruction.

    Entry into the blood-stream, through, for example, cuts, abrasions or lesions, may produce systemic injury with harmful effects. Examine
    the skin prior to the use of the material and ensure that any external damage is suitably protected.
    INHALED

    The material can cause respiratory irritation in some persons. The body's response to such irritation can cause further lung damage.

    Corrosive acids can cause irritation of the respiratory tract, with coughing, choking and mucous membrane damage. There may be
    dizziness, headache, nausea and weakness. Swelling of the lungs can occur, either immediately or after a delay; symptoms of this include
    chest tightness, shortness of breath, frothy phlegm and cyanosis. Lack of oxygen can cause death hours after onset.

    Inhalation of chlorosilane vapours or hydrochloric acid vapours or mist may cause damage of the respiratory tract. Chlorosilane injury from
    inhalation primarily affects the upper respiratory tract, causing inflammation, oedema and corrosive burns of the oral, nasal and pharyngeal
    mucosa and the upper airways.

    The material has NOT been classified as "harmful by inhalation". This is because of the lack of corroborating animal or human evidence. In
    the absence of such evidence, care should nevertheless be taken to ensure exposure is kept to a minimum and that suitable control
    measures be used, in an occupational setting to control vapors, fumes and aerosols.

    Inhalation hazard is increased at higher temperatures.

    Inhalation of quantities of liquid mist may be extremely hazardous, even lethal due to spasm, extreme irritation of larynx and bronchi,
    chemical pneumonitis and pulmonary edema.

    Hydrogen chloride (HCl) vapour or fumes present a hazard from a single acute exposure. Exposures of 1300 to 2000 ppm have been lethal
    to humans in a few minutes.
    Inhalation of HCl may cause choking, coughing, burning sensation and may  cause ulceration of the nose, throat and larynx. Fluid on the
    lungs followed by generalised lung damage may follow.
    Breathing of HCl vapour may aggravate asthma and inflammatory or fibrotic pulmonary disease.
    High concentrations cause necrosis of the tracheal and bronchial epithelium, pulmonary oedema, atelectasis and emphysema and damage

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